By Cordelia Fine

ISBN-10: 0393331636

ISBN-13: 9780393331639

The brain's strength is proven and touted on a daily basis in new experiences and examine. And but we have a tendency to take our brains without any consideration, with no suspecting that these lots of hard-working neurons will possibly not consistently be operating for us. Cordelia advantageous introduces us to a mind we would no longer are looking to meet, a mind with a brain of its personal. She illustrates the brain's tendency towards self-delusion as she explores how the brain defends and glorifies the ego through twisting and warping our perceptions. Our brains hire a slew of inborn mind-bugs and prejudices, from hindsight bias to unrealistic optimism, from ethical excuse-making to wishful thinking—all designed to avoid us from seeing the reality concerning the global and the folks round us, and approximately ourselves.

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Extra info for A Mind of Its Own: How Your Brain Distorts and Deceives

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A few transmission-disequilibrium studies of genes in this region, including GABRB3, have yielded nominally positive findings (Cook et al. 1998), but the findings from several others have been negative, as expected in a heterogeneous synMolecular Neurobiology of Childhood-Onset Psychiatric Disorders 21 drome and when a marker rather than a functional variant is studied. 00022. A number of as yet unreplicated family-based associations have been noted in other genes in autism, including HOXA1, AVPR1A, RELN, and WNT2.

The coloboma mutation is a chromosomal deletion encompassing a number of genes, including snap, which encodes SNAP-25, a synaptic vesicle docking protein that appears to be particularly important in presynaptic regulation of dopamine and norepinephrine release (Jones et al. 2001). When snap is transgenically returned to this mouse lineage, the mice return to normal levels of activity (Hess et al. 1996). An inbred rat model is the most extensively studied animal model for ADHD. Spontaneously hypertensive rats (SHRs), bred to manifest hypertension, also demonstrate marked hyperactivity as juveniles that decreases with administration of methylMolecular Neurobiology of Childhood-Onset Psychiatric Disorders 11 phenidate or D-amphetamine (Sagvolden et al.

2002). When hoarding was considered as a dichotomous trait, no significant evidence for linkage was detected. The candidate gene approach has not generated significant association findings in childhood-onset OCD; although association studies have implicated a number of genes in adult-onset OCD, including HTR1B, COMT, HTT, and MAOA, that have not yet been examined in childhood-onset samples. Eating Disorders Central nervous system neurochemistry and genetics have dominated research on the molecular neurobiology of eating disorders.

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A Mind of Its Own: How Your Brain Distorts and Deceives by Cordelia Fine

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